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- W2073279008 abstract "The G-protein-coupled estrogen receptor-1 (GPER, formerly known as GPR30) has attracted increasing interest, considering its ability to mediate estrogenic signaling in different cell types, including the hormone-sensitive tumors like breast cancer. As observed for other GPCR-mediated responses, the activation of the epidermal growth factor receptor is a fundamental integration point in the biological action triggered by GPER. A wide number of natural and synthetic compounds, including estrogens and anti-estrogens, elicit stimulatory effects in breast cancer through GPER up-regulation and activation, suggesting that GPER function is associated with breast tumor progression and tamoxifen resistance. GPER has also been proposed as a candidate biomarker in triple-negative breast cancer, opening a novel scenario for a more comprehensive assessment of breast tumor patients." @default.
- W2073279008 created "2016-06-24" @default.
- W2073279008 creator A5012914217 @default.
- W2073279008 creator A5046533203 @default.
- W2073279008 date "2014-05-06" @default.
- W2073279008 modified "2023-10-15" @default.
- W2073279008 title "GPER Function in Breast Cancer: An Overview" @default.
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- W2073279008 doi "https://doi.org/10.3389/fendo.2014.00066" @default.
- W2073279008 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4018520" @default.
- W2073279008 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24834064" @default.
- W2073279008 hasPublicationYear "2014" @default.
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