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- W2073451673 abstract "Reactive oxygen species (ROS)-induced damage on host cells and molecules has been considered the most likely proximal mechanism responsible for the age-related decline in organismal performance. Organisms have two possible ways to reduce the negative effect of ROS: disposing of effective antioxidant defenses and minimizing ROS production. The unbalance between the amount of ROS produced and the availability of antioxidant defenses determines the intensity of so-called oxidative stress. Interestingly, most studies that deal with the effect of oxidative stress on organismal performance have focused on the antioxidant defense compartment and, surprisingly, have neglected the mechanisms that control ROS production within mitochondria. Uncoupling proteins (UCPs), mitochondrial transporters of the inner membrane, are involved in the control of redox state of cells and in the production of mitochondrial ROS. Given their function, UCPs might therefore represent a major mechanistic link between metabolic activity and fitness. We suggest that by exploring the role of expression and function of UCPs both in experimental as well as in comparative studies, evolutionary biologists may gain better insight into this link." @default.
- W2073451673 created "2016-06-24" @default.
- W2073451673 creator A5004962893 @default.
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- W2073451673 creator A5063066225 @default.
- W2073451673 creator A5080327662 @default.
- W2073451673 date "2005-12-01" @default.
- W2073451673 modified "2023-10-12" @default.
- W2073451673 title "Mitochondrial Uncoupling Proteins: New Perspectives for Evolutionary Ecologists" @default.
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