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• W2073574675 abstract "Abstract 1. A sucrose gap system was used to record action potentials and mechanical responses of flounder heart. 2. Diltiazem eliminated mechanical responses and strongly inhibited the action potential plateau while nifedipine only slightly reduced cardiac contractions without significantly changing the action potential. 3. Verapamil slightly hyperpolarized flounder heart but was without effect on either the action potential or mechanical activity except at very high concentrations. 4. Lanthanum was ineffective at 2 mM on flounder heart, but manganese at 3 mM substantially inhibited electrical and mechanical responses accompanied by a small hyperpolarization. Substitution of manganese for calcium abolished all flounder cardiac activity. 5. BAY K 8644 enhanced cardiac force and enhanced the action potential plateau while depolarizing the preparations. Calcium-free salines abolished heart contractions and the action potential plateau while the spike phase persisted. 6. Low sodium salines enhanced while sodium-free salines abolished all heart activity as did tetrodotoxin above I μM. Tetrodotoxin abolished the action potential spike leaving only a small plateau phase. 7. Substituting lithium for sodium hyperpolarized the heart, enhanced contractions and prolonged the action potential plateau. Ouabain enhanced cardiac activity and depolarized the heart but ferosemide was without effect on either electrical or mechanical activity. 8. TEA at 6 mM had a modest positive inotropic effect and negative chronotropic effect on the heart while the action potential plateau phase was enhanced. 9. These results indicate that extracellular sodium and calcium are crucial in flounder heart electrogenesis but such a major role for potassium could not be established." @default.
• W2073574675 created "2016-06-24" @default.
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• W2073574675 date "1989-01-01" @default.
• W2073574675 modified "2023-09-27" @default.
• W2073574675 title "Electrophysiology of the flounder heart (Platichthys flesus)—the effect of agents which modify transmembrane ion transport" @default.
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• W2073574675 doi "https://doi.org/10.1016/0742-8413(89)90104-7" @default.
• W2073574675 hasPublicationYear "1989" @default.
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