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- W2074849382 endingPage "197" @default.
- W2074849382 startingPage "183" @default.
- W2074849382 abstract "The classical BCR–ABL negative myeloproliferative neoplasms (MPN) polycythemia vera, essential thrombocythemia, and primary myelofibrosis are clonal hematopoietic disorders characterized by excessive production of terminally differentiated myeloid cells. In MPN patients, the disease can progress to secondary myelofibrosis or acute myeloid leukemia. Clonal hematopoiesis, disease phenotype, and progression are caused by somatically acquired genetic lesions of genes involved in cytokine signaling, RNA splicing, as well as epigenetic regulation. This review provides an overview of point mutations and cytogenetic lesions associated with MPN and addresses the role of these somatic lesions in MPN disease progression." @default.
- W2074849382 created "2016-06-24" @default.
- W2074849382 creator A5003768458 @default.
- W2074849382 creator A5007091163 @default.
- W2074849382 date "2012-12-12" @default.
- W2074849382 modified "2023-09-25" @default.
- W2074849382 title "Genetic and epigenetic alterations of myeloproliferative disorders" @default.
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