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- W2075733118 abstract "It is concluded that the defect in Tay-Sachs disease is for an enzyme degrading a monosialoganglioside, in Niemann-Pick disease for an enzyme degrading sphingomyelin or possibly ceramide, in chronic Gaucher舗s disease for an enzyme degrading a glucocerebroside, and in metachromatic leucodystrophy for an enzyme for degradation of sulfatide. Alzheimer舗s disease does not appear to involve any specific changes in lipid composition. An hypothesis to explain the findings in Alzheimer舗s disease is presented." @default.
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- W2075733118 date "1965-05-01" @default.
- W2075733118 modified "2023-09-24" @default.
- W2075733118 title "Speculations on the nature of the metabolic defects in Tay-Sachs, Niemann-Pick, Gaucher舗s and Alzheimer舗s diseases, and metachromatic leucodystrophy" @default.
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- W2075733118 doi "https://doi.org/10.1007/bf02635578" @default.
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