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- W2076234413 abstract "Epidemiological, physiological and molecular models of colon carcinogenesis have been proposed. Consistent epidemiological risk factors include reduced plant-food intake (increased risk); elevated meat intake (increased risk); higher physical activity (reduced risk); and increased alcohol intake (increased risk). At the physiological level, these lifestyle variables may trigger processes that provide explanations for the associations: higher meat, fat and alcohol means more heterocyclic amines and higher levels of bile acids; higher plant food means higher intake of several anticarcinogens and fibre fermentation that produces volatile fatty acids; exercise has a variety of beneficial effects. This complexity is elaborated further in the context of the colonie milieu where interactions among digesta, bacteria and epithelial cells occur. The long-term likelihood of cancer is the summation of moment-to-moment changes in the colonie milieu brought about by this interaction. Possible relationships between established epidemiological risk factors, genetic susceptibility and somatic genetic changes are outlined." @default.
- W2076234413 created "2016-06-24" @default.
- W2076234413 creator A5048819576 @default.
- W2076234413 date "1995-07-01" @default.
- W2076234413 modified "2023-09-23" @default.
- W2076234413 title "Risk factors for colon neoplasia—epidemiology and biology" @default.
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- W2076234413 doi "https://doi.org/10.1016/0959-8049(95)00125-3" @default.
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