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• W2076552336 startingPage "2812" @default.
• W2076552336 abstract "Mammalian life span can be extended by both calorie restriction (CR) and mutations that diminish somatotropic signaling. Sirt1 is a mediator of many effects of CR in mammals, but any role in controlling somatotropic signaling has not been shown. Since the somatotropic axis is controlled by the brain, we created mice lacking Sirt1 specifically in the brain and examined the impacts of this manipulation on somatotropic signaling and the CR response. These mutant mice displayed defects in somatotropic signaling when fed ad libitum, and defects in the endocrine and behavioral responses to CR. We conclude that Sirt1 in the brain is a link between somatotropic signaling and CR in mammals." @default.
• W2076552336 created "2016-06-24" @default.
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• W2076552336 date "2009-12-15" @default.
• W2076552336 modified "2023-09-23" @default.
• W2076552336 title "Neuronal SIRT1 regulates endocrine and behavioral responses to calorie restriction" @default.
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• W2076552336 doi "https://doi.org/10.1101/gad.1839209" @default.
• W2076552336 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2827846" @default.
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