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• W2076702091 endingPage "7932" @default.
• W2076702091 startingPage "7927" @default.
• W2076702091 abstract "ABSTRACT Infection with Chlamydia pneumoniae has been implicated as a potential risk factor for atherosclerosis. This study demonstrated the effects of gamma interferon (IFN-γ)-mediated indoleamine 2,3-dioxygenase activity on C. pneumoniae persistence in HEp-2 cells, inclusion morphology, and ultrastructure. C. pneumoniae replication showed a dose-dependent decrease when treated with increasing concentrations of IFN-γ and a phenotypic switch resulting in a decrease in typical inclusions with an increase in smaller, less-dense atypical inclusions. Ultrastructural analysis of IFN-γ-treated C. pneumoniae revealed atypical inclusions containing large reticulatate-like aberrant bodies with no evidence of redifferentiation into elementary bodies." @default.
• W2076702091 created "2016-06-24" @default.
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• W2076702091 date "2001-12-01" @default.
• W2076702091 modified "2023-09-23" @default.
• W2076702091 title "Characterization of<i>Chlamydia pneumoniae</i>Persistence in HEp-2 Cells Treated with Gamma Interferon" @default.
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• W2076702091 doi "https://doi.org/10.1128/iai.69.12.7927-7932.2001" @default.
• W2076702091 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/98893" @default.
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