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- W2076907265 abstract "A strong causal link between increased adipose tissue mass and insulin resistance in tissues such as liver and skeletal muscle exists in obesity-related disorders such as type 2 diabetes. Increased adipose tissue mass in obese patients and patients with diabetes is associated with altered secretion of adipokines, which also includes chemotactic proteins. Adipose tissue releases a wide range of chemotactic proteins including many chemokines and chemerin, which are interesting targets for adipose tissue biology and for biomedical research in obesity and obesity-related diseases. This class of adipokines may be directly linked to a chronic state of low-grade inflammation and macrophage infiltration in adipose tissue, a concept intensively studied in adipose tissue biology in recent years. The inflammatory state of adipose tissue in obese patients may be the most important factor linking increased adipose tissue mass to insulin resistance. Furthermore, chemoattractant adipokines may play an important role in this situation, as many of these proteins possess biological activity beyond the recruitment of immune cells including effects on adipogenesis and glucose homeostasis in insulin-sensitive tissues. The present review provides a summary of experimental evidence of the role of adipose tissue-derived chemotactic cytokines and their function in insulin resistance in vivo and in vitro." @default.
- W2076907265 created "2016-06-24" @default.
- W2076907265 creator A5020607761 @default.
- W2076907265 creator A5031987417 @default.
- W2076907265 date "2009-08-24" @default.
- W2076907265 modified "2023-10-18" @default.
- W2076907265 title "Chemotactic cytokines, obesity and type 2 diabetes:<i>in vivo</i>and<i>in vitro</i>evidence for a possible causal correlation?" @default.
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- W2076907265 doi "https://doi.org/10.1017/s0029665109990218" @default.
- W2076907265 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19698204" @default.
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