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- W2077202337 endingPage "F616" @default.
- W2077202337 startingPage "F606" @default.
- W2077202337 abstract "Recent evidence indicates that interstitial infiltration of T cells and macrophages plays a role in the pathogenesis of salt-sensitive hypertension. The present review examines this evidence and summarizes the investigations linking the renal accumulation of immune cells and oxidative stress in the development of hypertension. The mechanisms involved in the hypertensive effects of oxidant stress and tubulointerstitial inflammation, in particular intrarenal ANG II activity, are discussed, focusing on their potential for sodium retention. The possibility of autoimmune reactivity in hypertension is raised in the light of the proinflammatory and immunogenic pathways stimulated by the interrelationship between oxidant stress and inflammatory response. Finally, we present some clinical considerations derived from the recognition of this interrelationship." @default.
- W2077202337 created "2016-06-24" @default.
- W2077202337 creator A5004471228 @default.
- W2077202337 creator A5026153184 @default.
- W2077202337 creator A5030266408 @default.
- W2077202337 creator A5056238061 @default.
- W2077202337 date "2004-04-01" @default.
- W2077202337 modified "2023-10-18" @default.
- W2077202337 title "Oxidative stress, renal infiltration of immune cells, and salt-sensitive hypertension: all for one and one for all" @default.
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