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- W2077264192 endingPage "e0122057" @default.
- W2077264192 startingPage "e0122057" @default.
- W2077264192 abstract "The type 3 adenylyl cyclase (AC3) is localized to olfactory cilia in the main olfactory epithelium (MOE) and primary cilia in the adult mouse brain. Although AC3 has been strongly implicated in odor perception and olfactory sensory neuron (OSN) targeting, its role in granule cells (GCs), the most abundant interneurons in the main olfactory bulb (MOB), remains largely unknown. Here, we report that the deletion of AC3 leads to a significant reduction in the size of the MOB as well as the level of adult neurogenesis. The cell proliferation and cell cycle in the subventricular zone (SVZ), however, are not suppressed in AC3−/− mice. Furthermore, AC3 deletion elevates the apoptosis of GCs and disrupts the maturation of newly formed GCs. Collectively, our results identify a fundamental role for AC3 in the development of adult-born GCs in the MOB." @default.
- W2077264192 created "2016-06-24" @default.
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- W2077264192 creator A5053098849 @default.
- W2077264192 creator A5060234326 @default.
- W2077264192 creator A5070991841 @default.
- W2077264192 date "2015-03-25" @default.
- W2077264192 modified "2023-10-01" @default.
- W2077264192 title "The Type 3 Adenylyl Cyclase Is Required for the Survival and Maturation of Newly Generated Granule Cells in the Olfactory Bulb" @default.
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