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- W2077374442 endingPage "612" @default.
- W2077374442 startingPage "586" @default.
- W2077374442 abstract "Human immunodeficiency virus (HIV) infection is principally a mucosal disease and the gastrointestinal (GI) tract is the major site of HIV replication. Loss of CD4+ T cells and systemic immune hyperactivation are the hallmarks of HIV infection. The end of acute infection is associated with the emergence of specific CD4+ and CD8+ T cell responses and the establishment of a chronic phase of infection. Abnormal levels of immune activation and inflammation persist despite a low steady state level of viremia. Although the causes of persistent immune hyperactivation remain incompletely characterized, physiological alterations of gastrointestinal tract probably play a major role. Failure to restore Th17 cells in gut-associated lymphoid tissues (GALT) might impair the recovery of the gut mucosal barrier. This review discusses recent advances on understanding the contribution of CD4+ T cell depletion to HIV pathogenesis." @default.
- W2077374442 created "2016-06-24" @default.
- W2077374442 creator A5017744326 @default.
- W2077374442 creator A5052658996 @default.
- W2077374442 creator A5077539950 @default.
- W2077374442 date "2011-05-12" @default.
- W2077374442 modified "2023-10-04" @default.
- W2077374442 title "CD4+ T Cell Depletion in Human Immunodeficiency Virus (HIV) Infection: Role of Apoptosis" @default.
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