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- W2077444875 endingPage "1336" @default.
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- W2077444875 abstract "Lambert-Eaton myasthenic syndrome is a rare, but reasonably well-understood, antibody-mediated autoimmune disease that is caused by serum auto-antibodies and results in muscle weakness and autonomic dysfunction. One half of the patients have an idiopathic form, the other half a tumour-associated form of the disease. Three randomised trials and a large number of smaller clinical studies have resulted in a number of drugs becoming available for the treatment of Lambert-Eaton myasthenic syndrome. Several drugs are available for the symptomatic treatment of the disease, including guanidine, aminopyridines or acetylcholinesterase inhibitors. Other therapies aim to deplete the serum autoantibodies or to suppress the immune system. For this purpose, immunomodulating strategies, such as intravenous immunoglobulins or plasmapheresis, or several immunosuppressive agents are available. Chemotherapy has successfully ameliorated the course of disease in Lambert-Eaton myasthenic syndrome patients with an underlying tumour." @default.
- W2077444875 created "2016-06-24" @default.
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- W2077444875 date "2006-06-29" @default.
- W2077444875 modified "2023-09-23" @default.
- W2077444875 title "Available treatment options for the management of Lambert-Eaton myasthenic syndrome" @default.
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- W2077444875 doi "https://doi.org/10.1517/14656566.7.10.1323" @default.
- W2077444875 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16805718" @default.
- W2077444875 hasPublicationYear "2006" @default.
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