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- W2077486579 endingPage "744" @default.
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- W2077486579 abstract "Autoimmune uveitis is a complex group of sight-threatening diseases that arise without a known infectious trigger. The disorder is often associated with immunological responses to retinal proteins. Experimental models of autoimmune uveitis targeting retinal proteins have led to a better understanding of the basic immunological mechanisms involved in the pathogenesis of uveitis and have provided a template for the development of novel therapies. The disease in humans is believed to be T cell-dependent, as clinical uveitis is ameliorated by T cell-targeting therapies. The roles of T helper 1 (Th1) and Th17 cells have been major topics of interest in the past decade. Studies in uveitis patients and experiments in animal models have revealed that Th1 and Th17 cells can both be pathogenic effectors, although, paradoxically, some cytokines produced by these subsets can also be protective, depending on when and where they are produced. The major proinflammatory as well as regulatory cytokines in uveitis, the therapeutic approaches, and benefits of targeting these cytokines will be discussed in this review." @default.
- W2077486579 created "2016-06-24" @default.
- W2077486579 creator A5004732672 @default.
- W2077486579 creator A5037231222 @default.
- W2077486579 date "2011-10-01" @default.
- W2077486579 modified "2023-09-26" @default.
- W2077486579 title "Cytokines in Autoimmune Uveitis" @default.
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- W2077486579 doi "https://doi.org/10.1089/jir.2011.0042" @default.