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- W2077486952 abstract "Prolonged hemodynamic overload results in cardiac hypertrophy with detrimental changes in myocardial gene expression and morphology that predict morbidity and mortality. Stretch sensors, necessary for myocyte enlargement, are thought to become impaired as a result of prolonged overload. However, the mechanisms by which chronic overload leads to failure are poorly understood. Cardiac muscle LIM protein (MLP) is thought to be a mechanosensor, possibly relaying signals from the cytoskeleton to the nucleus. Therefore the subcellular location of myocytic MLP may have important functional implications in health and disease." @default.
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- W2077486952 date "2006-06-01" @default.
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- W2077486952 title "A47. Decreased levels of non-nuclear muscle LIM protein are associated with cardiac dysfunction and heart failure" @default.
- W2077486952 doi "https://doi.org/10.1016/j.yjmcc.2006.03.285" @default.
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