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• W2077520123 abstract "Purely electrical heart diseases, defined by the absence of any structural cardiac defects, are responsible for a large number of sudden, unexpected deaths in otherwise healthy, young individuals. These conditions include the long QT syndrome, Brugada syndrome, catecholaminergic polymorphic ventricular tachycardia and the short QT syndrome. Collectively, these conditions have been referred to as channelopathies. Ion channels provide the molecular basis for cardiac electrical activity. These channels have specific ion selectivity and are responsible for the precise and timely regulation of the passage of charged ions across the cell membrane in myocytes, and the summation of their activity in cardiac muscle defines the surface electrocardiogram. Impairment in the flow of these ions in heart cells may mean the difference between a normal, prosperous life and the tragedy of a sudden, unexpected death due to ventricular arrhythmia. The present paper reviews the current clinical and molecular understanding of the electrical diseases of the heart associated with sudden cardiac death." @default.
• W2077520123 created "2016-06-24" @default.
• W2077520123 creator A5045543779 @default.
• W2077520123 creator A5045710602 @default.
• W2077520123 date "2007-08-01" @default.
• W2077520123 modified "2023-09-25" @default.
• W2077520123 title "Electrical heart disease: Genetic and molecular basis of cardiac arrhythmias in normal structural hearts" @default.
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• W2077520123 doi "https://doi.org/10.1016/s0828-282x(07)71001-2" @default.
• W2077520123 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2786999" @default.
• W2077520123 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/17668083" @default.
• W2077520123 hasPublicationYear "2007" @default.
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