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• W2077855852 abstract "Various congenital anomalies of the eyelids can result from abnormal tissue proliferation, fusion, and reopening of the eyelids. Therefore, it is important to study the molecular mechanisms underlying eyelid development, focusing on cell behaviors. Mammalian eyelid development occurs in four steps: specification, growth, epithelial fusion, and reopening. Epithelial-mesenchymal interactions are fundamental processes during eyelid formation, and epithelial factors functioning within the eyelid epithelium are also critical. Studies with mutant and genetically modified mice have revealed that various signaling pathways and transcriptional factors are involved in mouse eyelid development. In this review, eyelid morphogenetic factors or pathways are described, as revealed by their mutant phenotype, eye-open at birth (EOB). These include FGFR2b-FGF10, EGFR-ERK, MEKK-JNK, BMP, Shh, Wnt, GPR48, Jun, Forkhead, and Grainyhead." @default.
• W2077855852 created "2016-06-24" @default.
• W2077855852 creator A5001842284 @default.
• W2077855852 date "2012-10-01" @default.
• W2077855852 modified "2023-09-24" @default.
• W2077855852 title "Wakayama Symposium: Epithelial-Mesenchymal Interactions in Eyelid Development" @default.
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• W2077855852 doi "https://doi.org/10.1016/j.jtos.2012.07.005" @default.
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