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- W2077883552 endingPage "1089" @default.
- W2077883552 startingPage "1065" @default.
- W2077883552 abstract "Mitochondria contribute to cardiac dysfunction and myocyte injury via a loss of metabolic capacity and by the production and release of toxic products. This article discusses aspects of mitochondrial structure and metabolism that are pertinent to the role of mitochondria in cardiac disease. Generalized mechanisms of mitochondrial-derived myocyte injury are also discussed, as are the strengths and weaknesses of experimental models used to study the contribution of mitochondria to cardiac injury. Finally, the involvement of mitochondria in the pathogenesis of specific cardiac disease states (ischemia, reperfusion, aging, ischemic preconditioning, and cardiomyopathy) is addressed." @default.
- W2077883552 created "2016-06-24" @default.
- W2077883552 creator A5014524067 @default.
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- W2077883552 creator A5062897619 @default.
- W2077883552 creator A5063050485 @default.
- W2077883552 creator A5083025409 @default.
- W2077883552 date "2001-06-01" @default.
- W2077883552 modified "2023-10-15" @default.
- W2077883552 title "Mitochondrial Dysfunction in Cardiac Disease: Ischemia–Reperfusion, Aging, and Heart Failure" @default.
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