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- W2077954350 abstract "Chromosome 16 abnormalities occur mostly in acute myeloid leukemia (AML) French–American–British type M4, but occasionally in M5 and M2 [ [1] Campbell L.J. Challis J. Fok T. Garson O.M. Chromosome 16 abnormalities associated with myeloid malignancies. Genes Chromosomes Cancer. 1991; 3: 55-61 Crossref PubMed Scopus (45) Google Scholar ]. The most common abnormality is a pericentric inversion 16, which results in a fusion gene of 3′MYHII (myosin heavy chain gene at 16p13) and 5′CBFβ (core-binding factor β at 16q22). The inversion can be detected by fluorescence in situ hybridization (FISH) with probes of specific design. The earliest probe was a partial paint within the short arm of chromosome 16 (16p), designed so that a break in 16p would result in two signals of the same color on opposite sides of the centromere. More recent probes have been designed for the 16q locus as pull-apart probes in two colors, such that an inversion separates the two colors, again on opposite sides of the centromere. A straightforward inversion will be detected by using one of these probes [ 1 Campbell L.J. Challis J. Fok T. Garson O.M. Chromosome 16 abnormalities associated with myeloid malignancies. Genes Chromosomes Cancer. 1991; 3: 55-61 Crossref PubMed Scopus (45) Google Scholar , 2 Egan N. O'Reilly J. Chipper L. Higgins M. Herrmann R. Cannell P. Deletion of CBFB in a patient with acute myelomonocytic leukemia (AML M4Eo) and inversion 16. Cancer Genet Cytogenet. 2004; 154: 60-62 Abstract Full Text Full Text PDF PubMed Scopus (14) Google Scholar ]." @default.
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- W2077954350 date "2007-01-01" @default.
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- W2077954350 title "Deletion of 3′CBFβ in an inv(16)(p13.lq22) ascertained by fluorescence in situ hybridization and reverse-transcriptase polymerase chain reaction" @default.
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- W2077954350 doi "https://doi.org/10.1016/j.cancergencyto.2006.08.006" @default.
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