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• W2078140555 abstract "Abstract Trisomy 8 (+8), one of the most common chromosomal abnormalities found in patients with myelodysplastic syndromes ( MDS ), is occasionally seen in patients with otherwise typical aplastic anemia ( AA ). Although some studies have indicated that the presence of +8 is associated with the immune pathophysiology of bone marrow ( BM ) failure, its pathophysiology may be heterogeneous. We studied 53 patients (22 with AA and 31 with low‐risk MDS ) with +8 for the presence of increased glycosylphosphatidylinositol‐anchored protein‐deficient ( GPI ‐ AP − ) cells, their response to immunosuppressive therapy ( IST ), and their prognosis. A significant increase in the percentage of GPI ‐ AP − cells was found in 14 (26%) of the 53 patients. Of the 26 patients who received IST , including nine with increased GPI ‐ AP − cells and 17 without increased GPI ‐ AP − cells, 14 (88% with increased GPI ‐ AP − cells and 41% without increased GPI ‐ AP − cells) improved. The overall and event‐free survival rates of the +8 patients with and without increased GPI ‐ AP − cells at 5 yr were 100% and 100% and 59% and 57%, respectively. Examining the peripheral blood for the presence of increased GPI ‐ AP − cells may thus be helpful for choosing the optimal treatment for +8 patients with AA or low‐risk MDS ." @default.
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• W2078140555 date "2015-02-04" @default.
• W2078140555 modified "2023-10-03" @default.
• W2078140555 title "Increased glycosylphosphatidylinositol-anchored protein-deficient granulocytes define a benign subset of bone marrow failures in patients with trisomy 8" @default.
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• W2078140555 doi "https://doi.org/10.1111/ejh.12484" @default.
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