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- W207841909 endingPage "128" @default.
- W207841909 startingPage "121" @default.
- W207841909 abstract "Significant conservation in cognitive and neurodevelopmental mechanisms exist between Drosophila and mammals. In addition, while mechanisms of aging are relatively well conserved, Drosophila age much faster than mammals. Thus Drosophila are an ideal organism in which to study cognitive and neurodegenerative diseases. Powerful genetic techniques exist that allow genes to be disrupted and overexpressed easily. Phenotypes observed from these mutants can then be used to screen for genetic modifiers to elucidate pathways involved in disease pathology. Here we describe recent advances obtained from Drosophila models for five diseases, neurofibromatosis 1, fragile X syndrome, Alzheimer’s disease, tauopathies, and Parkinson’s disease." @default.
- W207841909 created "2016-06-24" @default.
- W207841909 creator A5015720701 @default.
- W207841909 creator A5019808324 @default.
- W207841909 date "2008-03-06" @default.
- W207841909 modified "2023-09-30" @default.
- W207841909 title "Modeling Cognitive and Neurodegenerative Disorders in Drosophila melanogaster" @default.
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- W207841909 doi "https://doi.org/10.1007/978-1-59745-285-4_15" @default.
- W207841909 hasPublicationYear "2008" @default.
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