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- W2078591587 abstract "Hypoglycaemia has been recognized as a clinical entity for only about fifty years, although some of the mechanisms by which it may be produced must have been operating throughout the ages. After the introduc tion of insulin for the treatment of diabetes in 1922 insulin reactions were observed for the first time, and Seale Harris then realized that he had seen patients with similar symptoms who were not being given insulin ; thus in 1924 he introduced the concept of spon taneous hypoglycaemia (Harris, 1924), and since then papers too numerous to quote have been devoted to the subject. I have used the term endogenous rather than spontaneous because it is more accurate, since there must always be a reason for the development of hypo glycaemia even if it cannot be fully understood, and endogenous hypoglycaemia may be defined as the produc tion of an abnormally low blood-sugar level by any means other than the exogenous administration of insulin, usually giving rise to symptoms or, less often, to physical signs. My observations have been limited to those patients in whom hypoglycaemia appeared to be the primary factor in the production of symptoms, thereby excluding hypoglycaemia which is part of a complex biochemical syndrome, as in gross liver disease or endocrine disorders and in other uncommon conditions. There remain for consideration three main types of endogenous hypo glycaemia?namely, organic hyperinsulinism resulting from the presence of functioning pancreatic islet-cell tumours, hypoglycaemia arising as a sequel to various gastric operations, and psychosomatic hypoglycaemia." @default.
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- W2078591587 date "1956-12-29" @default.
- W2078591587 modified "2023-09-26" @default.
- W2078591587 title "Some Neurological Manifestations of Endogenous Hypoglycaemia" @default.
- W2078591587 cites W1995770941 @default.
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- W2078591587 doi "https://doi.org/10.1136/bmj.2.5008.1512" @default.
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