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- W2078600808 abstract "See related article, pp 378–383Hypertension and cardiovascular diseases are more prevalent in men than in premenopausal women of the same age. However, after menopause, this cardiovascular protection in women is lost, possibly because of the unbalance of estrogen production and other sex hormones in postmenopause. Although the mechanisms underlying these sex differences remain unclear, divergences in the function of the renin-angiotensin system (RAS) and differences in the response to stimulation and inhibition of RAS in men and premenopausal women have been proposed.1,2RAS is involved in blood pressure regulation by modulating vascular tone and renal excretory function. In particular, the kidney is involved in the long-term regulation of arterial pressure by modulating sodium excretion.1It is now generally accepted that RAS organization is dual and that beside the well-known main pressor axis (angiotensin-converting enzyme/angiotensin [Ang] II/Ang type 1 receptor [AT1R]), there is a second depressor protective axis consisting of Ang type 2 receptor (AT2R), angiotensin-converting enzyme 2, Ang-(1–7), and MasR.1 In particular, AT2R mediates the vasodilatory and natriuretic actions of angiotensin peptides. AT2R is expressed throughout the kidney in both vascular and tubular elements, and it is greatly expressed in renal proximal tubule cells.1,2 AT2R induces vasodilation in both resistance and capacitance vessels, protects …" @default.
- W2078600808 created "2016-06-24" @default.
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- W2078600808 date "2014-08-01" @default.
- W2078600808 modified "2023-09-25" @default.
- W2078600808 title "Impact of the Direct Angiotensin II Type 2 Receptor Stimulation on Renal Function" @default.
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- W2078600808 doi "https://doi.org/10.1161/hypertensionaha.114.03199" @default.
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