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- W2078698928 abstract "Recent studies have allowed a better understanding of the biology of restenosis. Neointimal thickening--also referred to as neointimal hyperplasia--occurs in response to experimental arterial injury. This process involves different steps which include smooth muscle cell activation, proliferation, and migration, and the production of extracellular matrix. Neointimal thickening has been identified as one of the mechanisms of restenosis after balloon angioplasty in humans. The factors which control neointimal thickening include growth factors, hormonal factors, and mechanical factors. Delinquent reendothelialization has been shown to have a permissive impact on smooth muscle cell proliferation. In addition to neointimal thickening, arterial remodeling also plays a major role in restenosis. Studies performed on animals and in human subjects have established the potential for constrictive remodeling to reduce the vessel lumen after angioplasty. Restenosis thus appears as a multifactorial entity that may be addressed in the future by a combined mechanical and pharmacological approach." @default.
- W2078698928 created "2016-06-24" @default.
- W2078698928 creator A5035928746 @default.
- W2078698928 creator A5058992043 @default.
- W2078698928 date "1997-09-01" @default.
- W2078698928 modified "2023-09-25" @default.
- W2078698928 title "The biology of restenosis" @default.
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