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• W2078769892 abstract "LSD and BOL (0.125–0.5 mg/kg) were equipotent in increasing the in vivo tyrosine hydroxylation in the striatum as measured by the accumulation of DOPA after inhibition of neuronal decarboxylase. However, with 2–4 mg/kg doses, the maximum effect of BOL was larger than that of LSD. LSD and BOL antagonized the apomorphine-induced decrease of DOPA accumulation, without affecting the haloperidol-induced increase. LSD like apomorphine inhibited the increase of DOPA accumulation seen after reserpine, cerebral hemisection and after γ-butyrolactone (GBL). The effect of apomorphine in rats given GBL was blocked by haloperidol, but not by BOL and promethazine, whereas that of LSD was inhibited by haloperidol, BOL, and promethazine. These findings suggest that LSD and BOL directly affect nigro-neostriatal dopamine neurons. LSD therefore appears to be a partial agonist and BOL a pure autoreceptors. It is proposed in addition that LSD activates and BOL blocks 5-HT receptors that control DOPA formation." @default.
• W2078769892 created "2016-06-24" @default.
• W2078769892 creator A5013418979 @default.
• W2078769892 date "1977-05-01" @default.
• W2078769892 modified "2023-09-26" @default.
• W2078769892 title "The effect of LSD and 2-bromo LSD on the striatal DOPA accumulation after decarboxylase inhibition in rats" @default.
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• W2078769892 doi "https://doi.org/10.1016/0014-2999(77)90162-5" @default.
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