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• W2078934464 abstract "Bone undergoes a continuous cycle of remodeling for maintenance and healing. For almost a decade it has been appreciated that the immune system is intricately linked to bone homeostasis. Both acute and chronic inflammatory responses have been shown to impact bone health. A common form of inflammatory disease that causes bone destruction is the chronic infectious disease known as periodontitis (PD). PD is a bacteria-driven inflammation of the tooth-supporting apparatus that leads to resorption of the alveolar (jaw) bone, often leading to tooth loss. At the host-bacteria interface, Toll-like receptors (TLRs) play an instructive role in the development of innate and T cell adaptive responses to oral bacteria. Specifically, it is becoming apparent that TLR2-mediated inflammatory responses represent the major arm of the host immune response during periodontitis, and form an important link between periodontal infection and ensuing periodontal bone loss. This review summarizes the role of TLR2-mediated interplay between immune cells and bone cells in a periodontal disease setting." @default.
• W2078934464 created "2016-06-24" @default.
• W2078934464 creator A5060322860 @default.
• W2078934464 creator A5067750047 @default.
• W2078934464 creator A5090175941 @default.
• W2078934464 date "2013-09-05" @default.
• W2078934464 modified "2023-10-16" @default.
• W2078934464 title "Bacteria take control of tolls and T cells to destruct jaw bone" @default.
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• W2078934464 doi "https://doi.org/10.3109/08820139.2013.822761" @default.
• W2078934464 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24004056" @default.
• W2078934464 hasPublicationYear "2013" @default.
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