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• W2078981375 abstract "Acute kidney injury (AKI) is a rapidly developing condition often associated with critical illness, with a high degree of morbidity and mortality, whose pathophysiology is ill understood. Recent investigations have identified the dysfunction of the renal microcirculation and its cellular and subcellular constituents as being central to the etiology of AKI. Injury is caused by inflammatory activation involving endothelial leucocyte interactions in combination with dysregulation of the homeostatis between oxygen, nitric oxide, and reactive oxygen species. Effective therapies expected to resolve AKI will have to control inflammation and restore this homeostasis. In order to apply and guide these therapies effectively, diagnostic tools aimed at physiological biomarkers of AKI for monitoring renal microcirculatory function in advance of changes in pharmacological biomarkers associated with structural damage of the kidney will need to be developed." @default.
• W2078981375 created "2016-06-24" @default.
• W2078981375 creator A5057342507 @default.
• W2078981375 date "2014-09-24" @default.
• W2078981375 modified "2023-10-02" @default.
• W2078981375 title "The Central Role of Renal Microcirculatory Dysfunction in the Pathogenesis of Acute Kidney Injury" @default.
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• W2078981375 doi "https://doi.org/10.1159/000363203" @default.
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