FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W2079016614> ?p ?o ?g. }

• W2079016614 endingPage "80" @default.
• W2079016614 startingPage "67" @default.
• W2079016614 abstract "Degradation of proteins via the ubiquitin system is an important step in many stress signaling pathways in plants. E3 ligases recognize ligand proteins and dictate the high specificity of protein degradation, and thus, play a pivotal role in ubiquitination. Here, we identified a gene, named Arabidopsis thaliana abscisic acid (ABA)-insensitive RING protein 4 (AtAIRP4), which is induced by ABA and other stress treatments. AtAIRP4 encodes a cellular protein with a C3HC4-RING finger domain in its C-terminal side, which has in vitro E3 ligase activity. Loss of AtAIRP4 leads to a decrease in sensitivity of root elongation and stomatal closure to ABA, whereas overexpression of this gene in the T-DNA insertion mutant atairp4 effectively recovered the ABA-associated phenotypes. AtAIRP4 overexpression plants were hypersensitive to salt and osmotic stresses during seed germination, and showed drought avoidance compared with the wild-type and atairp4 mutant plants. In addition, the expression levels of ABA- and drought-induced marker genes in AtAIRP4 overexpression plants were markedly higher than those in the wild-type and atairp4 mutant plants. Hence, these results indicate that AtAIRP4 may act as a positive regulator of ABA-mediated drought avoidance and a negative regulator of salt tolerance in Arabidopsis." @default.
• W2079016614 created "2016-06-24" @default.
• W2079016614 creator A5003533766 @default.
• W2079016614 creator A5010577284 @default.
• W2079016614 creator A5018915097 @default.
• W2079016614 creator A5045468337 @default.
• W2079016614 creator A5056326728 @default.
• W2079016614 creator A5062430868 @default.
• W2079016614 creator A5090632160 @default.
• W2079016614 date "2015-11-19" @default.
• W2079016614 modified "2023-10-16" @default.
• W2079016614 title "<i>Arabidopsis</i> C3HC4-RING finger E3 ubiquitin ligase AtAIRP4 positively regulates stress-responsive abscisic acid signaling" @default.
• W2079016614 cites W1862289373 @default.
• W2079016614 cites W1963843265 @default.
• W2079016614 cites W1965737639 @default.
• W2079016614 cites W1973220227 @default.
• W2079016614 cites W1975110054 @default.
• W2079016614 cites W1983470920 @default.
• W2079016614 cites W1983505890 @default.
• W2079016614 cites W1984824352 @default.
• W2079016614 cites W1993171576 @default.
• W2079016614 cites W1995677293 @default.
• W2079016614 cites W2002900187 @default.
• W2079016614 cites W2006628865 @default.
• W2079016614 cites W2012683495 @default.
• W2079016614 cites W2019383594 @default.
• W2079016614 cites W2027692179 @default.
• W2079016614 cites W2029382359 @default.
• W2079016614 cites W2029690199 @default.
• W2079016614 cites W2039456164 @default.
• W2079016614 cites W2044536026 @default.
• W2079016614 cites W2047708521 @default.
• W2079016614 cites W2052698203 @default.
• W2079016614 cites W2052821301 @default.
• W2079016614 cites W2057122564 @default.
• W2079016614 cites W2057165823 @default.
• W2079016614 cites W2064766598 @default.
• W2079016614 cites W2066299394 @default.
• W2079016614 cites W2078823122 @default.
• W2079016614 cites W2088811537 @default.
• W2079016614 cites W2089259913 @default.
• W2079016614 cites W2095811247 @default.
• W2079016614 cites W2097590249 @default.
• W2079016614 cites W2097895495 @default.
• W2079016614 cites W2098115340 @default.
• W2079016614 cites W2099157613 @default.
• W2079016614 cites W2100945610 @default.
• W2079016614 cites W2104362188 @default.
• W2079016614 cites W2106541825 @default.
• W2079016614 cites W2106596862 @default.
• W2079016614 cites W2109202455 @default.
• W2079016614 cites W2110311890 @default.
• W2079016614 cites W2112713711 @default.
• W2079016614 cites W2115195173 @default.
• W2079016614 cites W2115242242 @default.
• W2079016614 cites W2116122104 @default.
• W2079016614 cites W2116352973 @default.
• W2079016614 cites W2125121305 @default.
• W2079016614 cites W2138032440 @default.
• W2079016614 cites W2142704168 @default.
• W2079016614 cites W2143148026 @default.
• W2079016614 cites W2146719438 @default.
• W2079016614 cites W2149098862 @default.
• W2079016614 cites W2151852675 @default.
• W2079016614 cites W2152972832 @default.
• W2079016614 cites W2154117244 @default.
• W2079016614 cites W2154368075 @default.
• W2079016614 cites W2154614604 @default.
• W2079016614 cites W2156231007 @default.
• W2079016614 cites W2157236929 @default.
• W2079016614 cites W2165902959 @default.
• W2079016614 cites W2167179803 @default.
• W2079016614 doi "https://doi.org/10.1111/jipb.12364" @default.
• W2079016614 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25913143" @default.
• W2079016614 hasPublicationYear "2015" @default.
• W2079016614 type Work @default.
• W2079016614 sameAs 2079016614 @default.
• W2079016614 citedByCount "51" @default.
• W2079016614 countsByYear W20790166142016 @default.
• W2079016614 countsByYear W20790166142017 @default.
• W2079016614 countsByYear W20790166142018 @default.
• W2079016614 countsByYear W20790166142019 @default.
• W2079016614 countsByYear W20790166142020 @default.
• W2079016614 countsByYear W20790166142021 @default.
• W2079016614 countsByYear W20790166142022 @default.
• W2079016614 countsByYear W20790166142023 @default.
• W2079016614 crossrefType "journal-article" @default.
• W2079016614 hasAuthorship W2079016614A5003533766 @default.
• W2079016614 hasAuthorship W2079016614A5010577284 @default.
• W2079016614 hasAuthorship W2079016614A5018915097 @default.
• W2079016614 hasAuthorship W2079016614A5045468337 @default.
• W2079016614 hasAuthorship W2079016614A5056326728 @default.
• W2079016614 hasAuthorship W2079016614A5062430868 @default.
• W2079016614 hasAuthorship W2079016614A5090632160 @default.
• W2079016614 hasBestOaLocation W20790166141 @default.
• W2079016614 hasConcept C104317684 @default.
• W2079016614 hasConcept C134459356 @default.
• W2079016614 hasConcept C143065580 @default.

• next