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- W2079108727 endingPage "1377" @default.
- W2079108727 startingPage "1367" @default.
- W2079108727 abstract "Studies of mechanisms of resistance to glyphosate have increased current understanding of herbicide resistance mechanisms. Thus far, single-codon non-synonymous mutations of EPSPS (5-enolypyruvylshikimate-3-phosphate synthase) have been rare and, relative to other herbicide mode of action target-site mutations, unconventionally weak in magnitude for resistance to glyphosate. However, it is possible that weeds will emerge with non-synonymous mutations of two codons of EPSPS to produce an enzyme endowing greater resistance to glyphosate. Today, target-gene duplication is a common glyphosate resistance mechanism and could become a fundamental process for developing any resistance trait. Based on competition and substrate selectivity studies in several species, rapid vacuole sequestration of glyphosate occurs via a transporter mechanism. Conversely, as the chloroplast requires transporters for uptake of important metabolites, transporters associated with the two plastid membranes may separately, or together, successfully block glyphosate delivery. A model based on finite glyphosate dose and limiting time required for chloroplast loading sets the stage for understanding how uniquely different mechanisms can contribute to overall glyphosate resistance." @default.
- W2079108727 created "2016-06-24" @default.
- W2079108727 creator A5040758372 @default.
- W2079108727 creator A5080533862 @default.
- W2079108727 date "2014-03-12" @default.
- W2079108727 modified "2023-10-14" @default.
- W2079108727 title "Glyphosate resistance: state of knowledge" @default.
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