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- W2079360957 abstract "The transcriptional activator IRF-1 and its antagonistic repressor IRF-2 are regulators of the interferon (IFN) system and of cell growth. Overexpression of IRF-2 leads to transformation of NIH3T3 cells, and the concomitant overexpression of IRF-1 reverts this transformed phenotype. Here we report that c-myc- or fosB-transformed rat embryonic fibroblast cells can be reverted by the introduction of the IRF-1 gene. Thus, the anti-oncogenic function of IRF-1 is not limited to only IRF-2 overexpressing cells, suggesting the broad role of IRF-1 as a tumor suppressor." @default.
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- W2079360957 date "1994-08-01" @default.
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- W2079360957 title "Suppression of c-myc or fosB-induced cell transformation by the transcription factor IRF-1" @default.
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- W2079360957 doi "https://doi.org/10.1016/0304-3835(94)90318-2" @default.
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