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• W2079657156 abstract "The type I interferon (IFN) system plays a pivotal role in the etiopathogenesis of systemic lupus erythematosus (SLE). The initial appearance of autoantibody-producing B cells can be precipitated by infection-induced type I IFNs, but the further, significant generation of autoimmune T and B cells is caused by the prolonged production of IFN-alpha, which is maintained by a vicious circle mechanism. This involves the activation of immature dendritic cells, known as natural IFN-producing cells, by continuously formed endogenous IFN-alpha inducers. These IFN-alpha inducers consist of complexes of autoantibodies with nucleic-acid-containing autoantigens derived from apoptotic cells." @default.
• W2079657156 created "2016-06-24" @default.
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• W2079657156 date "2001-08-01" @default.
• W2079657156 modified "2023-10-13" @default.
• W2079657156 title "An etiopathogenic role for the type I IFN system in SLE" @default.
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• W2079657156 doi "https://doi.org/10.1016/s1471-4906(01)01955-x" @default.
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