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• W2079706925 abstract "The proapoptotic cytokine TRAIL has been shown to enhance amyloid-beta-dependent neurotoxicity. Here are reported interactions between TRAIL and nitric oxide (NO) in cultured rat astrocytes in vitro. Rat astrocytes expressed all TRAIL receptor mRNAs and proteins. However, TRAIL failed in inducing apoptosis of astrocytes, whereas these cells released substantial amounts of nitrites. A TRAIL-neutralizing antibody was able to prevent LPS-induced iNOS expression in astrocytes. Interestingly, TRAIL induced its own expression in astrocytes. These data suggest that redundancy between TRAIL and NO in astrocytes could be fueling neuronal damage/death processes, potentially uncovering novel molecular targets for the treatment of neurodegenerative disorders." @default.
• W2079706925 created "2016-06-24" @default.
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• W2079706925 date "2007-01-01" @default.
• W2079706925 modified "2023-09-23" @default.
• W2079706925 title "Trail interacts redundantly with nitric oxide in rat astrocytes: Potential contribution to neurodegenerative processes" @default.
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• W2079706925 doi "https://doi.org/10.1016/j.jneuroim.2006.09.007" @default.
• W2079706925 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/17067687" @default.
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