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• W2079710612 endingPage "1354" @default.
• W2079710612 startingPage "1345" @default.
• W2079710612 abstract "The aim of the study was to investigate the inhibitory control of an ongoing motor response and to identify underlying neural deficiencies, manifested in event-related potentials, that cause poorer inhibitory performance in children with attention-deficit/hyperactivity disorder.A stop-signal paradigm with a primary visual task and auditory stop signal was used to compare performance in 13 children with attention-deficit/hyperactivity disorder and 13 control children, while event-related potentials were recorded simultaneously.Children with attention-deficit/hyperactivity disorder showed poorer inhibitory performance through a slower inhibitory process. Inhibitory processing of auditory stop signals was marked by a frontal N2 component that was reduced in the attention-deficit/hyperactivity disorder group relative to controls. A central positive component (P3) was associated with the success of inhibiting a response, but there were no group differences in its amplitude or latency.Findings support the hypothesis of deficient inhibitory control in children with attention-deficit/hyperactivity disorder. Slower inhibitory processing appears to be due to a specific neural deficiency that manifests in the processing of the stop signal as attenuated negativity in the N2 latency range." @default.
• W2079710612 created "2016-06-24" @default.
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• W2079710612 date "2003-12-01" @default.
• W2079710612 modified "2023-09-30" @default.
• W2079710612 title "Inhibitory motor control in children with attention-deficit/hyperactivity disorder: event-related potentials in the stop-signal paradigm" @default.
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• W2079710612 doi "https://doi.org/10.1016/s0006-3223(03)00703-0" @default.
• W2079710612 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/14675798" @default.
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