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• W2079719673 startingPage "255" @default.
• W2079719673 abstract "Although safe and effective vaccines for hepatitis B virus (HBV) have been available for nearly three decades, this virus kills at least 600,000 people annually worldwide and remains the leading global cause of chronic hepatitis, cirrhosis, and hepatocellular carcinoma. Because the HBV reverse transcriptase lacks a proofreading function, many HBV genotypes, subgenotypes, mutants, and recombinants exist. At least 10 HBV genotypes (HBV-A through J) with distinct geographic distributions have been identified; by definition, their complete genomic sequences diverge by more than 8%. HBV genotype is increasingly becoming recognized as an important factor in the progression and clinical outcome of HBV-induced disease. Infections by HBV-C or -D are significantly more likely to lead to cirrhosis and hepatocellular carcinoma than are infections by HBV-A or -B. Additionally, the hepatitis B e antigen seroconversion response to standard or pegylated interferon is more favorable in patients with HBV-A or -B than in those with HBV-C or -D. However, therapeutic responses to nucleos(t)ide analogues are generally comparable among HBV genotypes. In conclusion, genotyping of HBV is useful in identifying chronic hepatitis B patients who are at increased risk of disease progression, thereby enabling physicians to optimize antiviral therapy for these patients." @default.
• W2079719673 created "2016-06-24" @default.
• W2079719673 creator A5063550464 @default.
• W2079719673 date "2011-01-01" @default.
• W2079719673 modified "2023-10-18" @default.
• W2079719673 title "Molecular Epidemiology of Hepatitis B Virus" @default.
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• W2079719673 doi "https://doi.org/10.3904/kjim.2011.26.3.255" @default.
• W2079719673 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3192197" @default.
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