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- W2079745823 endingPage "678" @default.
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- W2079745823 abstract "Cholesterol is an important precursor for numerous biologically active molecules, and it plays a major role in membrane structure and function. Cholesterol can be endogenously synthesized or exogenously taken up via the endocytic vesicle system and subsequently delivered to post-endo/lysosomal sites including the plasma membrane and the endoplasmic reticulum. Niemann–Pick C (NPC) disease results in the accumulation of exogenously-derived cholesterol, as well as other lipids, in late endosomes and lysosomes (LE/LY). Identification of the two genes that underlie NPC disease, NPC1 and NPC2, has focused attention on the mechanisms by which lipids, in particular cholesterol, are transported out of the LE/LY compartment. This review discusses the role of the NPC2 protein in cholesterol transport, and the potential for concerted action of NPC1 and NPC2 in regulating normal intracellular cholesterol homeostasis." @default.
- W2079745823 created "2016-06-24" @default.
- W2079745823 creator A5044773512 @default.
- W2079745823 creator A5045014224 @default.
- W2079745823 date "2009-07-01" @default.
- W2079745823 modified "2023-10-12" @default.
- W2079745823 title "Niemann–Pick C2 (NPC2) and intracellular cholesterol trafficking" @default.
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