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• W2079771431 abstract "Technological advances in antibody generation and production have facilitated recent clinical and commercial success with antibody-based cancer therapeutics. The class III receptor tyrosine kinase FLT3 is highly expressed on the blast cells in most cases of acute myelogenous leukemia (AML) and B-cell acute lymphoblastic leukemia (ALL). Activating mutations of FLT3 are detected in approximately 37% AML patients. FLT3 expression in normal tissue is limited to myeloid and B-cell precursor cells. Therefore, over-expressed or mutated FLT3 is an attractive target for therapeutic intervention using monoclonal antibodies. This review will discuss recent progress in the development of anti-FLT3 antibodies as well as their therapeutic potentials in the treatment of AML and other hematological malignancies. Drug Dev. Res. 67:495–500, 2006. © 2006 Wiley-Liss, Inc." @default.
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• W2079771431 date "2006-01-01" @default.
• W2079771431 modified "2023-09-27" @default.
• W2079771431 title "FLT3 Antibody-based therapy for leukemia" @default.
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• W2079771431 doi "https://doi.org/10.1002/ddr.20112" @default.
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