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- W2079777658 abstract "The incretin hormones glucagon-like peptide (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP) are secreted by the intestine in response to nutrient intake. Incretins have a variety of metabolic effects such as signalling the pancreas to increase insulin and reduce glucagon secretion and thereby inhibiting hepatic glucose production [ [1] Holst J.J. The physiology of glucagon-like peptide 1. Physiol. Rev. 2007; 87: 1409-1439 Crossref PubMed Scopus (2172) Google Scholar ]. In healthy individuals, incretins are thought to be responsible for up to 70% of the prandial insulin response; however, the incretin effect is impaired in individuals with type 2 diabetes [ [2] Nauck M. Stöckmann F. Ebert R. Creutzfeldt W. Reduced incretin effect in type 2 (non-insulin-dependent) diabetes. Diabetologia. 1986; 29: 46-52 Crossref PubMed Scopus (1073) Google Scholar ]." @default.
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- W2079777658 date "2009-07-01" @default.
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- W2079777658 title "GLP-1 receptor agonists: Targeting both hyperglycaemia and disease processes in diabetes" @default.
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- W2079777658 doi "https://doi.org/10.1016/j.diabres.2009.02.017" @default.
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