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• W2079786621 abstract "Pregnancy is characterized by a complex interplay of inflammatory events regulated by both the innate and acquired immune systems. Similarly, parturition can be viewed as the activation of “pro-labour” inflammatory pathways, which drive cervical ripening and myometrial activation. Premature activation of these pathways, for example, by infection, can lead to preterm labour and birth. Nuclear factor κβ is a key modulator of these pathways and functions by regulating the expression of prostaglandins, chemokines and pro-inflammatory cytokines involved in both term and preterm labour. Future design of therapeutics that target key mediators of inflammation and immune activation would therefore be a rational approach for preventing preterm labour and immune-mediated neonatal brain damage." @default.
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• W2079786621 date "2012-03-13" @default.
• W2079786621 modified "2023-10-01" @default.
• W2079786621 title "Prevention of preterm labour via the modulation of inflammatory pathways" @default.
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• W2079786621 doi "https://doi.org/10.3109/14767058.2012.666114" @default.
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