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- W2079984893 abstract "The long-standing free radical theory of aging, which attributes cellular pathology to the relentless accumulation of reactive oxygen species (ROS), remains attractive but controversial. Emerging insights into the molecular interactions between ROS and reactive nitrogen species (RNS) such as nitric oxide suggest that, in biological systems, one effect of increased ROS is the disruption of protein S-nitrosylation, a ubiquitous posttranslational modification system. In this way, ROS may not only damage cells but also disrupt widespread signaling pathways. Here, we discuss this phenomenon in the context of the cardiovascular system and propose that ideas regarding oxidative stress and aging need to be reevaluated to take account of the balance between oxidative and nitrosative stress." @default.
- W2079984893 created "2016-06-24" @default.
- W2079984893 creator A5047554076 @default.
- W2079984893 creator A5060026327 @default.
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- W2079984893 date "2005-05-25" @default.
- W2079984893 modified "2023-10-16" @default.
- W2079984893 title "Nitric Oxide and Oxidative Stress in Cardiovascular Aging" @default.
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- W2079984893 doi "https://doi.org/10.1126/sageke.2005.21.re4" @default.
- W2079984893 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/15917507" @default.
- W2079984893 hasPublicationYear "2005" @default.
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