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• W2079986242 abstract "ATM and ATR are stress-response kinases which respond to a variety of insults including ionizing radiation, replication arrest, ultraviolet radiation and hypoxia/re-oxygenation. Hypoxia occupies a unique niche in the study of both ATR- and ATM-mediated checkpoint pathways. Hypoxia is a physiologically significant stress that occurs in virtually all solid tumors and differs from most other stresses in that it does not induce DNA damage. Previous studies have indicated that hypoxia provides a unique way to induce ATR in response to inhibition of DNA replication. During tumor expansion hypoxia is inevitably followed by periods of re-oxygenation which in vitro has been shown to induce significant levels of DNA damage and an ATM response. Therefore both ATR and ATM have a role to play in hypoxia/re-oxygenation." @default.
• W2079986242 created "2016-06-24" @default.
• W2079986242 creator A5018081801 @default.
• W2079986242 creator A5080172191 @default.
• W2079986242 date "2004-08-01" @default.
• W2079986242 modified "2023-10-14" @default.
• W2079986242 title "The role of ATM and ATR in the cellular response to hypoxia and re-oxygenation" @default.
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• W2079986242 doi "https://doi.org/10.1016/j.dnarep.2004.03.035" @default.
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