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• W2080229178 abstract "We studied cerebral perforating arteries in 18 autopsied cases of cerebral infarction, using monoclonal antibodies, antiactin antibody HHF 35, and antimacrophage antibody HAM 56. Degeneration and focal necrosis of smooth muscle cells were in the form of initial arterial lesions in patients with cerebral infarctions. These lesions were mostly detected in the outer layers of the media, and eventually developed into widespread necrosis. Following the progression of the medial changes, monocytes, possibly migrating from Virchow-Robin's space, accumulated in the adventitia. In advanced lesions, monocytes in the blood stream penetrated through the endothelial cells and accumulated in the subendothelial space and the media. Monocytes migrating from Virchow-Robin's space, as well as from the blood stream, thickened the arterial wall and narrowed the lumen, while accelerating fibrosis and plasma deposition. Applying immunocytochemical techniques we note that monocytes, HAM 56 positive cells, may have played a leading role in the development of occlusive arterial lesions, resulting in cerebral infarction." @default.
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• W2080229178 date "1993-01-01" @default.
• W2080229178 modified "2023-09-26" @default.
• W2080229178 title "Immunocytochemical study of cerebral perforating arteries in patients with cerebral infarctions." @default.
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• W2080229178 doi "https://doi.org/10.1267/ahc.26.109" @default.
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