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- W2080443362 abstract "Plasmodium falciparum malaria parasites invade human red blood cells and immediately begin making significant alterations to the structure of the erythrocyte. These alterations facilitate the movement of nutrients into, and waste products and parasite-derived proteins out of the cell to meet the needs of the growing parasite. A tubovesicular membrane network extending from the parasite vacuole membrane probably has a central role in the transport processes. The parasite also modifies the erythrocyte membrane itself in a way that not only changes its permeablilty but also places parasite-derived proteins in knob-like protrusions at the cell surface. These proteins enable the parasite to adhere to endothelial cells and thereby avoid clearance from the blood stream by the spleen. Antigenic variation of these proteins allows parasitized erythrocytes to vary their phenotype and produce a sustained and chronic malaria infection. Study of the molecular processes that underlie these parasite-induced modifications of the host red blood cell will lead to improved understanding of malaria pathogenesis and, perhaps, suggest new approaches against the disease." @default.
- W2080443362 created "2016-06-24" @default.
- W2080443362 creator A5017226115 @default.
- W2080443362 creator A5067549205 @default.
- W2080443362 date "1996-02-01" @default.
- W2080443362 modified "2023-09-23" @default.
- W2080443362 title "Membrane modifications in erythrocytes parasitized by Plasmodium falciparum" @default.
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- W2080443362 doi "https://doi.org/10.1016/0166-6851(95)02575-8" @default.
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