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- W2080777321 abstract "Received May 6, 2003; revision received June 11, 2003; accepted June 19, 2003. From the Department of Psychiatry, Laval Hospital. Address reprint requests to Dr. Sirois, Department of Psychiatry, Laval Hospital, 2725, chemin Sainte-Foy, Sainte-Foy, Quebec, Canada G1V 4G5; fsir@ globetrotter.net (e-mail). Copyright 2004 The Academy of Psychosomatic Medicine. Nephrogenic diabetes insipidus is a well-established complication of long-term lithium therapy. It may affect about 10%–15% of patients treated 15 years or more. It is known that long-term administration of lithium induces an irreversible reduction of urinary-concentrating capacity and a partly reversible reduction in the glomerular filtration rate. Such complications are usually heralded either by clinical symptoms of polydipsia, polyuria, and nocturia or by abnormal laboratory values of serum creatinine or urinary density. The onset of nephrogenic diabetes insipidus can be associated with a surgical episode in which it presents a challenge for fluid management. A previous report indicated that such an occurrence could have been suspected on clinical grounds had a exhaustive history been possible. The current report also emphasizes that a symptom of polydipsia was present; it does not stress the medical management of nephrogenic diabetes insipidus but opens the question of setting long-term lithium treatment short of significant renal impairment." @default.
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- W2080777321 date "2004-03-01" @default.
- W2080777321 modified "2023-09-25" @default.
- W2080777321 title "Lithium-Induced Nephrogenic Diabetes Insipidus in a Surgical Patient" @default.
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- W2080777321 doi "https://doi.org/10.1176/appi.psy.45.1.82" @default.
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