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- W2080836319 abstract "Parkinson's disease (PD) is a major health problem affecting millions of people worldwide. Recent studies provide compelling evidence that altered Ca(2) (+) homeostasis may underlie disease pathomechanism and be an inherent feature of all vulnerable neurons. The downstream effects of altered Ca(2) (+) handling in the distinct subcellular organelles for proper cellular function are beginning to be elucidated. Here, we summarize the evidence that vulnerable neurons may be exposed to homeostatic Ca(2) (+) stress which may determine their selective vulnerability, and suggest how abnormal Ca(2) (+) handling in the distinct intracellular compartments may compromise neuronal health in the context of aging, environmental, and genetic stress. Gaining a better understanding of the varied effects of Ca(2) (+) dyshomeostasis may allow novel combinatorial therapeutic strategies to slow PD progression." @default.
- W2080836319 created "2016-06-24" @default.
- W2080836319 creator A5034753362 @default.
- W2080836319 creator A5048609114 @default.
- W2080836319 creator A5063147284 @default.
- W2080836319 creator A5090189628 @default.
- W2080836319 date "2014-06-17" @default.
- W2080836319 modified "2023-10-16" @default.
- W2080836319 title "Upstream deregulation of calcium signaling in Parkinson’s disease" @default.
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