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- W208084154 abstract "This chapter discusses the role of receptors located in the left atrium of the heart or the pulmonal vascular bed in the short-term adjustment of sodium balance. The left atrium is a suitable place to measure an extracellular volume deficit. The extracellular volume can only be re-expanded if external sodium is available. Osmocontrol overrides volume control. Elevation of left atrial pressure reduces renin secretion by the kidneys. The renin–angiotensin–aldosterone system has two antinatriuretic components: a faster acting one, namely, AII, and a slower acting one, namely, aldosterone. Atrial natriuresis (AN) can be prevented by acute or chronic reduction of total body sodium. The RAS is more responsible for Na-retention than for Na-elimination, and cardiac denervation prevents AN, but does not disturb the postprandial adjustment of sodium balance." @default.
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- W208084154 date "1981-01-01" @default.
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- W208084154 title "IS THE CONTROL OF SODIUM EXCRETION PARTLY DUE TO SIGNALS FROM RECEPTORS LOCATED IN THE LEFT ATRIUM OF THE HEART?" @default.
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- W208084154 doi "https://doi.org/10.1016/b978-0-08-027339-6.50012-9" @default.
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