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- W2080847069 abstract "Many animal models of bacterial diseases are hampered by differences in tissue tropism and the course of pathogenesis. In a recent issue of Cell, by rationally mutating a surface invasion protein (InlA) to have higher binding affinity for its cognate host receptor (E-cadherin), Wollert et al. were able to murinizeListeria monocytogenes, creating a strain capable of invading intestinal epithelial cells in mice, mimicking the route of infection in humans." @default.
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- W2080847069 title "Designer Bugs: Structural Engineering to Build a Better Mouse Model" @default.
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- W2080847069 doi "https://doi.org/10.1016/j.chom.2007.05.009" @default.
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