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• W2082867657 abstract "We explored novel functions of tenascin-C by comparing mouse embryonic fibroblasts (MEFs) proficient or deficient in tenascin-C expression. Transcript profiling analysis identified tissue plasminogen activator (tPA) as the most consistently over-expressed gene in all tenascin-C deficient MEFs. This was confirmed by real-time PCR as well as by protein expression analysis. In agreement with these observations, tenascin-C deficient MEFs had an increased capacity to digest fibrin in situ. Consistently, tenascin-C expression in vivo was found to correlate with fibrin deposition in several diseases associated with tenascin-C overexpression such as fibrosis, asthma and cancer. In conclusion, the present study suggests a new role of tenascin-C as a regulator of the fibrinolytic system." @default.
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• W2082867657 date "2011-02-24" @default.
• W2082867657 modified "2023-10-17" @default.
• W2082867657 title "Tenascin-C triggers fibrin accumulation by downregulation of tissue plasminogen activator" @default.
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• W2082867657 doi "https://doi.org/10.1016/j.febslet.2011.02.023" @default.
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