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- W2083745793 abstract "Spinal cord injury initiates a complex series of inflammatory and immune responses including the influx of monocytes, macrophages, T‐cells, NK cells and so on, into the injured area. In the present study, we found a significant increase in the levels of granzyme‐b (gra‐b) from the first day after the transection until the third day, with decrease in intensity thereafter. The chemokine IP‐10/CXCL10 was also found to be elevated along with gra‐b correlating with the infiltration of CD‐8 + cytotoxic T lymphocytes (CTLs) into the injured spinal cord. We observed an increase in the levels of the 64 kDa poly ADP ribose polymerase fragment, known to be a signature fragment produced by gra‐b. Localization of gra‐b in TUNEL positive neurons indicates that gra‐b might play a crucial role in neuronal death and contributes to the pathophysiology of spinal cord injury." @default.
- W2083745793 created "2016-06-24" @default.
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- W2083745793 date "2009-05-13" @default.
- W2083745793 modified "2023-10-12" @default.
- W2083745793 title "Granzyme-b mediated cell death in the spinal cord-injured rat model" @default.
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- W2083745793 doi "https://doi.org/10.1111/j.1440-1789.2008.00980.x" @default.
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