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• W2084096055 abstract "N-Palmitoylethanolamide (PEA) is an endogenous lipid secreted by human adipocytes that possesses numerous anti-inflammatory properties. Human adipose tissue can be subjected to modulation of its inflammatory state by lipopolysaccharide (LPS). Here we demonstrate that LPS increases the secretion of interleukin-6 (IL-6) by human mature adipocytes via activation of the NFκB pathway. This effect is not inhibited by PEA. Inversely, LPS strongly inhibits adipose cell leptin release, with PEA acting as a potentiator of this inhibitory effect. These actions are not linked to a reduction in leptin gene transcription. Thus, PEA does not have an anti-inflammatory role in the secretion of IL-6 via NFκB at the adipocyte level, but instead seems to act at the heart of the LPS-stimulated pathway, which, independently of NFκB, inhibits the secretion of leptin." @default.
• W2084096055 created "2016-06-24" @default.
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• W2084096055 date "2006-06-01" @default.
• W2084096055 modified "2023-10-17" @default.
• W2084096055 title "Effect of PEA on LPS inflammatory action in human adipocytes" @default.
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• W2084096055 doi "https://doi.org/10.1016/j.cyto.2006.06.005" @default.
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