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- W2084164539 abstract "Glucocorticoid-induced osteoporosis is the most common cause of secondary osteoporosis. The role of the Wnt signaling pathway in bone formation and the ratio of receptor activator for NF-κB ligand versus osteoprotegerin in bone resorption are exciting new insights. The absolute fracture risk helps both clinicians and patients to interpret the results of bone density measurement, which may have a positive influence on adherence to therapy. The bisphosphonates alendronate and risedronate are the first-line treatment in the prevention of glucocorticoid-induced osteoporosis, because both increase the bone mineral density of the spine and hips and reduce the vertebral fracture rate. Treatment with the anabolic agent parathyroid hormone (1 – 34) strongly stimulates bone turnover, and seems to be superior to treatment with alendronate. It might be attractive for glucocorticoid-treated patients with new vertebral fractures during treatment with bisphosphonates, and/or with severe fracture risk." @default.
- W2084164539 created "2016-06-24" @default.
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- W2084164539 date "2009-04-01" @default.
- W2084164539 modified "2023-10-04" @default.
- W2084164539 title "Prevention of glucocorticoid-induced osteoporosis" @default.
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- W2084164539 doi "https://doi.org/10.1517/14656560902868225" @default.
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